Specijalizacija
Grad
Jezici
O doktoru
Prof. dr Đorđe Jevtović, rođen 1953. godine u Beogradu, lekar-infektolog. Profesor je Medicinskog fakulteta i načelnik centra za HIV na Klinici za infektologiju i tropske bolesti u Beogradu. Dr Jevtović je objavio 208 publikacija kojima su obuhvaćene različite oblasti infektologije.
Prof. dr Đorđe Jevtović je u penziji.
Poštovani profesore,
22 nakon nezaštićenog analnog odnosa (receptivni) sam uradio na Infektivnoj klinici u BG HIV antigen/antinody test. Negativan je. U trenutku testiranja sam imao i dalje imam, blago uvećane limfne žlezde na vratu kao i promenu boje koze spolja u tom predelu. Nemam febrilnost ali imam suvi kašalj, pušač sam. Takodje, hep B i C, negativni kao i ostale std. Da li je potrebno da ponovim testiranje na HIV? Hvala.
nema potrebe za daljim testiranjem
Doktore sta raditi dalje u vezi hpv da li je moguce posle toliko uradjenih pcr tesova sad pokaze u usnoj duplji samo pre mesec dsna ssm radio u gzzjz test usne duplje grla nepca jezika i bio negativan nikakve odnose nemam niti sam imao u medjuvremenu
pa nemate HPV infekciju, ako postoje ikakve promene nisu te etiologije
Hvala, Jos jedno pitanje: da li su moje vrednosti sa poslednjeg testa daleko, daleko od referentne vrednosti (broja) 1, 1.0, 1.00 ili 1.000 (kako-god se oznacava)?
važno e da je test negativan
Cut off index je 0.06. zabrinjavajuće bi bilo da je 0.6? Pitam kao laik. Zbog čega rastu, odnosno menjaju se COI vrednosti?
uvek je nula, s nekom nebitno decimalom.Testovi na HIV Ag/At bi trbalo izdavati kao pozitivan ili negativan, sve ostalo je nepotrebno
Doktore zvao sam neke labaratorije i oni su rekli kad je u stanju mirovanja kao herpes ne moze se otkriti ni pcr testom
OVO je iz udžbenika, sve drugo nije relevantno.Poređenje HSV i HPV je znak suštinskog nerazumevanja tih infekcija
Human papillomavirus (HPV) infects epithelial cells. Most of the > 100 subtypes infect cutaneous epithelium and cause skin warts; some types infect mucosal epithelium and cause anogenital warts. Skin or anogenital warts are diagnosed based on clinical appearance and are usually treated with topical medications or cytodestructive treatments. Genital warts may persist and spread widely in patients with decreased cell-mediated immunity (eg, due to pregnancy or HIV infection). Some types that infect mucosal epithelium can lead to anogenital or oropharyngeal cancer. Pap tests and/or HPV testing is recommended to screen for cervical cancer and anal cancer in high-risk patients. Vaccines are available to protect against many of the HPV strains that can cause genital warts and cancer.
HPV is the most common sexually transmitted infection (STI). HPV is so common that 80% of sexually active unvaccinated people get the virus at some point in their life (1). In the US, about 14 million people become newly infected with HPV each year; before the HPV vaccine became available, each year roughly 340,000 to 360,000 patients sought care for genital warts caused by HPV.
Most HPV infections clear spontaneously within 1 to 2 years, but some persist.
General reference
1. Centers for Disease Control and Prevention: Human papillomavirus (HPV). Accessed July 6, 2022.
Nucleic acid amplification tests (NAATs) for oncogenic HPV subtypes are used as part of routine cervical cancer screening in women. Initial tests typical detect any one of 13 common high-risk types. Follow-up HPV genotype tests may be done to detect the most high-risk types, typically 16, 18, or 45. No HPV testing for men is available for clinical use.
Clinicians should check for malignant oral lesions potentially caused by HPV during routine examination of the mouth and oral cavity.
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